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Tuberculosis and autoimmunity: Common features. / Starshinova, A.; Kudryavtsev, I.; Kudlay, D.; Zinchenko, Y.; Yablonskiy, P.

в: Tuberculosis, Том 134, 2022.

Результаты исследований: Научные публикации в периодических изданиях › статья › Рецензирование

Harvard

Starshinova, A, Kudryavtsev, I, Kudlay, D, Zinchenko, Y & Yablonskiy, P 2022, 'Tuberculosis and autoimmunity: Common features', Tuberculosis, Том. 134. https://doi.org/10.1016/j.tube.2022.102202

APA

Starshinova, A., Kudryavtsev, I., Kudlay, D., Zinchenko, Y., & Yablonskiy, P. (2022). Tuberculosis and autoimmunity: Common features. Tuberculosis, 134. https://doi.org/10.1016/j.tube.2022.102202

Vancouver

Starshinova A, Kudryavtsev I, Kudlay D, Zinchenko Y, Yablonskiy P. Tuberculosis and autoimmunity: Common features. Tuberculosis. 2022;134. https://doi.org/10.1016/j.tube.2022.102202

Author

Starshinova, A. ; Kudryavtsev, I. ; Kudlay, D. ; Zinchenko, Y. ; Yablonskiy, P. / Tuberculosis and autoimmunity: Common features. в: Tuberculosis. 2022 ; Том 134.

BibTeX

@article{7a2dde511952424492a38ac05ef2be42,
title = "Tuberculosis and autoimmunity: Common features",
abstract = "Despite the proven infectious nature of tuberculosis, new data have recently appeared on the autoimmune component in the course of tuberculosis infection. This chapter discusses the currently known signs of autoimmune inflammation in tuberculosis infection, including both clinical and immunological manifestations. Taking into account the available data on the possible triggering effect of M. tuberculosis on the development of the autoimmune process, the correction of these autoimmune complications may be a key moment in prescribing therapy and in predicting the low efficacy of the treatment of the disease. {\textcopyright} 2022 Elsevier Ltd",
keywords = "ASIA, Autoantibodies, Autoimmunity, HLA, Molecular mimicry, Tuberculosis, Autoimmune Diseases, Humans, Inflammation, Mycobacterium tuberculosis, HLA antigen, autoantibody, autoimmune disease, autoimmunity, B lymphocyte, genotype, helper cell, HLA system, human, immunology, meta analysis (topic), nonhuman, Review, T lymphocyte, Tfh cell, tuberculosis, complication, inflammation",
author = "A. Starshinova and I. Kudryavtsev and D. Kudlay and Y. Zinchenko and P. Yablonskiy",
note = "Export Date: 27 February 2026; Cited By: 27; Correspondence Address: A. Starshinova; Head of the Research Department, Almazov National Medical Research Centre, Saint-Petersburg, 2, Akkuratov str, 197341, Russian Federation; email: starshinova_aa@almazovcentre.ru; CODEN: TUBEC",
year = "2022",
doi = "10.1016/j.tube.2022.102202",
language = "Английский",
volume = "134",
journal = "Tuberculosis",
issn = "1472-9792",
publisher = "Churchill Livingstone",

}

RIS

TY - JOUR

T1 - Tuberculosis and autoimmunity: Common features

AU - Starshinova, A.

AU - Kudryavtsev, I.

AU - Kudlay, D.

AU - Zinchenko, Y.

AU - Yablonskiy, P.

N1 - Export Date: 27 February 2026; Cited By: 27; Correspondence Address: A. Starshinova; Head of the Research Department, Almazov National Medical Research Centre, Saint-Petersburg, 2, Akkuratov str, 197341, Russian Federation; email: starshinova_aa@almazovcentre.ru; CODEN: TUBEC

PY - 2022

Y1 - 2022

N2 - Despite the proven infectious nature of tuberculosis, new data have recently appeared on the autoimmune component in the course of tuberculosis infection. This chapter discusses the currently known signs of autoimmune inflammation in tuberculosis infection, including both clinical and immunological manifestations. Taking into account the available data on the possible triggering effect of M. tuberculosis on the development of the autoimmune process, the correction of these autoimmune complications may be a key moment in prescribing therapy and in predicting the low efficacy of the treatment of the disease. © 2022 Elsevier Ltd

AB - Despite the proven infectious nature of tuberculosis, new data have recently appeared on the autoimmune component in the course of tuberculosis infection. This chapter discusses the currently known signs of autoimmune inflammation in tuberculosis infection, including both clinical and immunological manifestations. Taking into account the available data on the possible triggering effect of M. tuberculosis on the development of the autoimmune process, the correction of these autoimmune complications may be a key moment in prescribing therapy and in predicting the low efficacy of the treatment of the disease. © 2022 Elsevier Ltd

KW - ASIA

KW - Autoantibodies

KW - Autoimmunity

KW - HLA

KW - Molecular mimicry

KW - Tuberculosis

KW - Autoimmune Diseases

KW - Humans

KW - Inflammation

KW - Mycobacterium tuberculosis

KW - HLA antigen

KW - autoantibody

KW - autoimmune disease

KW - autoimmunity

KW - B lymphocyte

KW - genotype

KW - helper cell

KW - HLA system

KW - human

KW - immunology

KW - meta analysis (topic)

KW - nonhuman

KW - Review

KW - T lymphocyte

KW - Tfh cell

KW - tuberculosis

KW - complication

KW - inflammation

U2 - 10.1016/j.tube.2022.102202

DO - 10.1016/j.tube.2022.102202

M3 - статья

VL - 134

JO - Tuberculosis

JF - Tuberculosis

SN - 1472-9792

ER -

ID: 149453971