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Mechanisms of cerebral damage in patients with HIV-infection. / Belyakov, N. A.; Medvedev, S. V.; Trofimova, T. N.; Rassokhln, V. V.; Dementeva, N. Ye; Shelomov, S. A.

в: Vestnik Rossiǐskoǐ akademii meditsinskikh nauk / Rossiǐskaia akademiia meditsinskikh nauk, № 9, 01.12.2012, стр. 4-12.

Результаты исследований: Научные публикации в периодических изданиях › статья › Рецензирование

Harvard

Belyakov, NA, Medvedev, SV, Trofimova, TN, Rassokhln, VV, Dementeva, NY & Shelomov, SA 2012, 'Mechanisms of cerebral damage in patients with HIV-infection', Vestnik Rossiǐskoǐ akademii meditsinskikh nauk / Rossiǐskaia akademiia meditsinskikh nauk, № 9, стр. 4-12.

APA

Belyakov, N. A., Medvedev, S. V., Trofimova, T. N., Rassokhln, V. V., Dementeva, N. Y., & Shelomov, S. A. (2012). Mechanisms of cerebral damage in patients with HIV-infection. Vestnik Rossiǐskoǐ akademii meditsinskikh nauk / Rossiǐskaia akademiia meditsinskikh nauk, (9), 4-12.

Vancouver

Belyakov NA, Medvedev SV, Trofimova TN, Rassokhln VV, Dementeva NY, Shelomov SA. Mechanisms of cerebral damage in patients with HIV-infection. Vestnik Rossiǐskoǐ akademii meditsinskikh nauk / Rossiǐskaia akademiia meditsinskikh nauk. 2012 Дек. 1;(9):4-12.

Author

Belyakov, N. A. ; Medvedev, S. V. ; Trofimova, T. N. ; Rassokhln, V. V. ; Dementeva, N. Ye ; Shelomov, S. A. / Mechanisms of cerebral damage in patients with HIV-infection. в: Vestnik Rossiǐskoǐ akademii meditsinskikh nauk / Rossiǐskaia akademiia meditsinskikh nauk. 2012 ; № 9. стр. 4-12.

BibTeX

@article{41e693fcb04b40ca9265aa378926cf4e,
title = "Mechanisms of cerebral damage in patients with HIV-infection",
abstract = "Mechanisms of HlV transportation through blood-brain barrier, vascular plexus and interaction with cerebral cells having CD-4-receptors, CCR-5- and CXCR-4-coreceptors were studied. Cerebral damage developed through latent and acute periods also known as HIV-encephalopathy, HIV-associated neurocognitive dysfunction etc. Cerebral lesions are caused by a variety of chemical agents from pro-inflammatory cytokines to toxic HIV-proteins resulting in development of HIV-dementia through several years. Even early stage of this process revealed significant disturbances of glucose metabolism and evoked potentials EEG alterations which can serve as a marker of HIV-infection. Genetic differences ofHWin blood and spinal liquor with different drug resistance were revealed implying a new approach to therapy development.",
keywords = "Brain, Cognitive dysfunction, HIV, Pathogenesis",
author = "Belyakov, {N. A.} and Medvedev, {S. V.} and Trofimova, {T. N.} and Rassokhln, {V. V.} and Dementeva, {N. Ye} and Shelomov, {S. A.}",
year = "2012",
month = dec,
day = "1",
language = "English",
pages = "4--12",
journal = "ВЕСТНИК РОССИЙСКОЙ АКАДЕМИИ МЕДИЦИНСКИХ НАУК",
issn = "0869-6047",
publisher = "Медицина",
number = "9",

}

RIS

TY - JOUR

T1 - Mechanisms of cerebral damage in patients with HIV-infection

AU - Belyakov, N. A.

AU - Medvedev, S. V.

AU - Trofimova, T. N.

AU - Rassokhln, V. V.

AU - Dementeva, N. Ye

AU - Shelomov, S. A.

PY - 2012/12/1

Y1 - 2012/12/1

N2 - Mechanisms of HlV transportation through blood-brain barrier, vascular plexus and interaction with cerebral cells having CD-4-receptors, CCR-5- and CXCR-4-coreceptors were studied. Cerebral damage developed through latent and acute periods also known as HIV-encephalopathy, HIV-associated neurocognitive dysfunction etc. Cerebral lesions are caused by a variety of chemical agents from pro-inflammatory cytokines to toxic HIV-proteins resulting in development of HIV-dementia through several years. Even early stage of this process revealed significant disturbances of glucose metabolism and evoked potentials EEG alterations which can serve as a marker of HIV-infection. Genetic differences ofHWin blood and spinal liquor with different drug resistance were revealed implying a new approach to therapy development.

AB - Mechanisms of HlV transportation through blood-brain barrier, vascular plexus and interaction with cerebral cells having CD-4-receptors, CCR-5- and CXCR-4-coreceptors were studied. Cerebral damage developed through latent and acute periods also known as HIV-encephalopathy, HIV-associated neurocognitive dysfunction etc. Cerebral lesions are caused by a variety of chemical agents from pro-inflammatory cytokines to toxic HIV-proteins resulting in development of HIV-dementia through several years. Even early stage of this process revealed significant disturbances of glucose metabolism and evoked potentials EEG alterations which can serve as a marker of HIV-infection. Genetic differences ofHWin blood and spinal liquor with different drug resistance were revealed implying a new approach to therapy development.

KW - Brain

KW - Cognitive dysfunction

KW - HIV

KW - Pathogenesis

UR - http://www.scopus.com/inward/record.url?scp=84872295073&partnerID=8YFLogxK

M3 - Article

C2 - 23210166

AN - SCOPUS:84872295073

SP - 4

EP - 12

JO - ВЕСТНИК РОССИЙСКОЙ АКАДЕМИИ МЕДИЦИНСКИХ НАУК

JF - ВЕСТНИК РОССИЙСКОЙ АКАДЕМИИ МЕДИЦИНСКИХ НАУК

SN - 0869-6047

IS - 9

ER -

ID: 34718087