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Evidance of amyloid nature of Aβ-GFP and PrP-GFP fusion proteins expressing in yeast. / Saifitdinova, Alsu; Rubel, Aleksandr; Lada, Artem; Vechtomov, Inge; Galkin, Aleksey.

в: Yeast, Том 24, № S1, S1, 27.06.2007, стр. S124.

Результаты исследований: Научные публикации в периодических изданияхтезисыРецензирование

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@article{9895eea6f5254d5eb994860988eed2e3,
title = "Evidance of amyloid nature of Aβ-GFP and PrP-GFP fusion proteins expressing in yeast",
abstract = "Baker's yeast S. cerevisiae being a genetically and functionally well-defined and easily handled eukaryotic cell is an attractive host for production of mammalian proteins and good tool for analysis of proteins misfolding and searching for therapeutic agents affecting amyloid aggregation and propagation. Fusion proteins Aβ-GFP and PrP-GFP overexpressed in yeast form oligomeres and high-weight aggregates as opposed to cells expressing bare GFP. Cytoplasmic localization of large fluorescent aggregates of the fusion proteins has been proved by staining with fluorescent dyes for particular cellular compartments. Fluorescence recovery after photobleaching (FRAP) examination of PrP-GFP fluorescent fibrils and visible clumps of Aβ-GFP in living cells have revealed significant portion of immobile fraction in the aggregates, which reflects tight stable interactions between individual molecules. These data are well agreed with high resistance of the fusion proteins to SDS treatment as well as yeast proteases and proteinase K digestion. The results suggest amyloid nature of PrP-GFP and Aβ-GFP aggregates in yeast making the system useful for analysis of factors affecting amyloidogenesis. This work is supported by Fogarty (TW006965-01A1), Ministry of Education and Science RF (PHII.2.2.2.3.10047) and CRDF (BRHE Y4-B-12-04).",
keywords = "дрожжи, амилоид бета, PrP",
author = "Alsu Saifitdinova and Aleksandr Rubel and Artem Lada and Inge Vechtomov and Aleksey Galkin",
note = "Saifitdinova A., Rubel A., Lada A., Inge-Vechtomov S., Galkin A. Evidance of amyloid nature of Aβ-GFP and PrP-GFP fusion proteins expressing in yeast // Materials of XXIII international confirence on yeast genetics and molecular biology.12. Yeast models for human disease and ageing. Yeast. - 2007. Vol 24, N S1 P. 125. https://doi.org/10.1002/yea.1529. ISSN 0749-503X.; null ; Conference date: 01-06-2007 Through 06-06-2007",
year = "2007",
month = jun,
day = "27",
doi = "https://doi.org/10.1002/yea.1529",
language = "English",
volume = "24",
pages = "S124",
journal = "Yeast",
issn = "0749-503X",
publisher = "Wiley-Blackwell",
number = "S1",
url = "http://www.yeast2007.org/",

}

RIS

TY - JOUR

T1 - Evidance of amyloid nature of Aβ-GFP and PrP-GFP fusion proteins expressing in yeast

AU - Saifitdinova, Alsu

AU - Rubel, Aleksandr

AU - Lada, Artem

AU - Vechtomov, Inge

AU - Galkin, Aleksey

N1 - Conference code: 23

PY - 2007/6/27

Y1 - 2007/6/27

N2 - Baker's yeast S. cerevisiae being a genetically and functionally well-defined and easily handled eukaryotic cell is an attractive host for production of mammalian proteins and good tool for analysis of proteins misfolding and searching for therapeutic agents affecting amyloid aggregation and propagation. Fusion proteins Aβ-GFP and PrP-GFP overexpressed in yeast form oligomeres and high-weight aggregates as opposed to cells expressing bare GFP. Cytoplasmic localization of large fluorescent aggregates of the fusion proteins has been proved by staining with fluorescent dyes for particular cellular compartments. Fluorescence recovery after photobleaching (FRAP) examination of PrP-GFP fluorescent fibrils and visible clumps of Aβ-GFP in living cells have revealed significant portion of immobile fraction in the aggregates, which reflects tight stable interactions between individual molecules. These data are well agreed with high resistance of the fusion proteins to SDS treatment as well as yeast proteases and proteinase K digestion. The results suggest amyloid nature of PrP-GFP and Aβ-GFP aggregates in yeast making the system useful for analysis of factors affecting amyloidogenesis. This work is supported by Fogarty (TW006965-01A1), Ministry of Education and Science RF (PHII.2.2.2.3.10047) and CRDF (BRHE Y4-B-12-04).

AB - Baker's yeast S. cerevisiae being a genetically and functionally well-defined and easily handled eukaryotic cell is an attractive host for production of mammalian proteins and good tool for analysis of proteins misfolding and searching for therapeutic agents affecting amyloid aggregation and propagation. Fusion proteins Aβ-GFP and PrP-GFP overexpressed in yeast form oligomeres and high-weight aggregates as opposed to cells expressing bare GFP. Cytoplasmic localization of large fluorescent aggregates of the fusion proteins has been proved by staining with fluorescent dyes for particular cellular compartments. Fluorescence recovery after photobleaching (FRAP) examination of PrP-GFP fluorescent fibrils and visible clumps of Aβ-GFP in living cells have revealed significant portion of immobile fraction in the aggregates, which reflects tight stable interactions between individual molecules. These data are well agreed with high resistance of the fusion proteins to SDS treatment as well as yeast proteases and proteinase K digestion. The results suggest amyloid nature of PrP-GFP and Aβ-GFP aggregates in yeast making the system useful for analysis of factors affecting amyloidogenesis. This work is supported by Fogarty (TW006965-01A1), Ministry of Education and Science RF (PHII.2.2.2.3.10047) and CRDF (BRHE Y4-B-12-04).

KW - дрожжи

KW - амилоид бета

KW - PrP

U2 - https://doi.org/10.1002/yea.1529

DO - https://doi.org/10.1002/yea.1529

M3 - Meeting Abstract

VL - 24

SP - S124

JO - Yeast

JF - Yeast

SN - 0749-503X

IS - S1

M1 - S1

Y2 - 1 June 2007 through 6 June 2007

ER -

ID: 99429650