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Deep Immunophenotyping of Circulating T and B Cells in Relapsing Adult-Onset Still's Disease. / Myachikova, Valentina; Kudryavtsev, Igor; Rubinstein, Artem; Aquino, Arthur; Isakov, Dmitry; Golovkin, Alexey; Maslyanskiy, Alexey.

в: Current Issues in Molecular Biology, Том 46, № 2, 01.02.2024, стр. 1177-1191.

Результаты исследований: Научные публикации в периодических изданияхстатьяРецензирование

Harvard

Myachikova, V, Kudryavtsev, I, Rubinstein, A, Aquino, A, Isakov, D, Golovkin, A & Maslyanskiy, A 2024, 'Deep Immunophenotyping of Circulating T and B Cells in Relapsing Adult-Onset Still's Disease', Current Issues in Molecular Biology, Том. 46, № 2, стр. 1177-1191. https://doi.org/10.3390/cimb46020075

APA

Myachikova, V., Kudryavtsev, I., Rubinstein, A., Aquino, A., Isakov, D., Golovkin, A., & Maslyanskiy, A. (2024). Deep Immunophenotyping of Circulating T and B Cells in Relapsing Adult-Onset Still's Disease. Current Issues in Molecular Biology, 46(2), 1177-1191. https://doi.org/10.3390/cimb46020075

Vancouver

Myachikova V, Kudryavtsev I, Rubinstein A, Aquino A, Isakov D, Golovkin A и пр. Deep Immunophenotyping of Circulating T and B Cells in Relapsing Adult-Onset Still's Disease. Current Issues in Molecular Biology. 2024 Февр. 1;46(2):1177-1191. https://doi.org/10.3390/cimb46020075

Author

Myachikova, Valentina ; Kudryavtsev, Igor ; Rubinstein, Artem ; Aquino, Arthur ; Isakov, Dmitry ; Golovkin, Alexey ; Maslyanskiy, Alexey. / Deep Immunophenotyping of Circulating T and B Cells in Relapsing Adult-Onset Still's Disease. в: Current Issues in Molecular Biology. 2024 ; Том 46, № 2. стр. 1177-1191.

BibTeX

@article{969e98f897684379aa1e65f5145975b1,
title = "Deep Immunophenotyping of Circulating T and B Cells in Relapsing Adult-Onset Still's Disease",
abstract = "Adult-onset Still's disease (AOSD) is a complex systemic inflammatory disorder, categorized as an 'IL-1 driven' inflammasomapathy. Despite this, the interaction between T and B cells remains poorly understood. We conducted a study, enrolling 7 patients with relapsing AOSD and 15 healthy control subjects, utilizing deep flow cytometry analysis to examine peripheral blood T- and B-cell subsets. T-cell and B-cell subsets were significantly altered in patients with AOSD. Within CD4+ T cells, Th2 cells were decreased. Additionally, Th17 cell and follicular Th cell subsets were altered within CD45RA-CD62L+ and CD45RA-CD62L- Th cells in patients with AOSD compared to healthy controls. We identified changes in CD8+ T cell maturation and 'polarization' in AOSD patients, with an elevated presence of the TEMRA CD8+ T cell subset. Furthermore, the percentage of Tc1 cells was decreased, while the frequency of CCR6-CXCR3- Tc2 cells was elevated. Finally, we determined that the frequency of CD5+CD27- B cells was dramatically decreased in patients with AOSD compared to healthy controls. Further investigations on a large group of patients with AOSD are required to evaluate these adaptive immunity cells in the disease pathogenesis.",
author = "Valentina Myachikova and Igor Kudryavtsev and Artem Rubinstein and Arthur Aquino and Dmitry Isakov and Alexey Golovkin and Alexey Maslyanskiy",
year = "2024",
month = feb,
day = "1",
doi = "10.3390/cimb46020075",
language = "English",
volume = "46",
pages = "1177--1191",
journal = "Current Issues in Molecular Biology",
issn = "1467-3037",
publisher = "Caister Academic Press",
number = "2",

}

RIS

TY - JOUR

T1 - Deep Immunophenotyping of Circulating T and B Cells in Relapsing Adult-Onset Still's Disease

AU - Myachikova, Valentina

AU - Kudryavtsev, Igor

AU - Rubinstein, Artem

AU - Aquino, Arthur

AU - Isakov, Dmitry

AU - Golovkin, Alexey

AU - Maslyanskiy, Alexey

PY - 2024/2/1

Y1 - 2024/2/1

N2 - Adult-onset Still's disease (AOSD) is a complex systemic inflammatory disorder, categorized as an 'IL-1 driven' inflammasomapathy. Despite this, the interaction between T and B cells remains poorly understood. We conducted a study, enrolling 7 patients with relapsing AOSD and 15 healthy control subjects, utilizing deep flow cytometry analysis to examine peripheral blood T- and B-cell subsets. T-cell and B-cell subsets were significantly altered in patients with AOSD. Within CD4+ T cells, Th2 cells were decreased. Additionally, Th17 cell and follicular Th cell subsets were altered within CD45RA-CD62L+ and CD45RA-CD62L- Th cells in patients with AOSD compared to healthy controls. We identified changes in CD8+ T cell maturation and 'polarization' in AOSD patients, with an elevated presence of the TEMRA CD8+ T cell subset. Furthermore, the percentage of Tc1 cells was decreased, while the frequency of CCR6-CXCR3- Tc2 cells was elevated. Finally, we determined that the frequency of CD5+CD27- B cells was dramatically decreased in patients with AOSD compared to healthy controls. Further investigations on a large group of patients with AOSD are required to evaluate these adaptive immunity cells in the disease pathogenesis.

AB - Adult-onset Still's disease (AOSD) is a complex systemic inflammatory disorder, categorized as an 'IL-1 driven' inflammasomapathy. Despite this, the interaction between T and B cells remains poorly understood. We conducted a study, enrolling 7 patients with relapsing AOSD and 15 healthy control subjects, utilizing deep flow cytometry analysis to examine peripheral blood T- and B-cell subsets. T-cell and B-cell subsets were significantly altered in patients with AOSD. Within CD4+ T cells, Th2 cells were decreased. Additionally, Th17 cell and follicular Th cell subsets were altered within CD45RA-CD62L+ and CD45RA-CD62L- Th cells in patients with AOSD compared to healthy controls. We identified changes in CD8+ T cell maturation and 'polarization' in AOSD patients, with an elevated presence of the TEMRA CD8+ T cell subset. Furthermore, the percentage of Tc1 cells was decreased, while the frequency of CCR6-CXCR3- Tc2 cells was elevated. Finally, we determined that the frequency of CD5+CD27- B cells was dramatically decreased in patients with AOSD compared to healthy controls. Further investigations on a large group of patients with AOSD are required to evaluate these adaptive immunity cells in the disease pathogenesis.

U2 - 10.3390/cimb46020075

DO - 10.3390/cimb46020075

M3 - Article

C2 - 38392193

VL - 46

SP - 1177

EP - 1191

JO - Current Issues in Molecular Biology

JF - Current Issues in Molecular Biology

SN - 1467-3037

IS - 2

ER -

ID: 117429786