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КЛИНИЧЕСКАЯ ПАТОФИЗИОЛОГИЯ РАННЕГО МЕТАБОЛИЧЕСКОГО СИНДРОМА: ПАТОГЕНЕТИЧЕСКАЯ РОЛЬ ЮНОШЕСКОГО ДИСПИТУИТАРИЗМА, ДИСПЛАЗИЙ СОЕДИНИТЕЛЬНОЙ ТКАНИ И АУТОИММУННОГО ТИРОИДИТА. / Stroev, I. I.; Churilov, L. P.; Agapov, M. M.; Kononova, I. A.; Mudzukova, O. M.; Sadov, S. A.; Serdiuk, I. I.

в: Patologicheskaia fiziologiia i èksperimental'naia terapiia, № 3, 01.07.2011, стр. 3-14.

Результаты исследований: Научные публикации в периодических изданияхОбзорная статьяРецензирование

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@article{8cfd867aad7b4a4b8bd3f72788d5f927,
title = "КЛИНИЧЕСКАЯ ПАТОФИЗИОЛОГИЯ РАННЕГО МЕТАБОЛИЧЕСКОГО СИНДРОМА:: ПАТОГЕНЕТИЧЕСКАЯ РОЛЬ ЮНОШЕСКОГО ДИСПИТУИТАРИЗМА, ДИСПЛАЗИЙ СОЕДИНИТЕЛЬНОЙ ТКАНИ И АУТОИММУННОГО ТИРОИДИТА",
abstract = "A review of world literature, including authors' original papers, establishes the clinical pathophysiological hypothesis, initially coined in by authors in 2002, about regular transition of adolescent hypothalamic syndrome (obesity with rose striae) with age into early metabolic syndrome, complicated by autoimmune thyroiditis. Some evidences are obtained, that witness for marfanoid phenotype and chronic disequilibrium between local, autacoid-mediated and systemic, hormone-mediated regulation, typical for inherited connective tissue disorders, may promote this transition (6 fig., 3 tables, bibl.: 92 ref.).",
author = "Stroev, {I. I.} and Churilov, {L. P.} and Agapov, {M. M.} and Kononova, {I. A.} and Mudzukova, {O. M.} and Sadov, {S. A.} and Serdiuk, {I. I.}",
year = "2011",
month = jul,
day = "1",
language = "русский",
pages = "3--14",
journal = "ПАТОЛОГИЧЕСКАЯ ФИЗИОЛОГИЯ И ЭКСПЕРИМЕНТАЛЬНАЯ ТЕРАПИЯ",
issn = "0031-2991",
publisher = "Медицина",
number = "3",

}

RIS

TY - JOUR

T1 - КЛИНИЧЕСКАЯ ПАТОФИЗИОЛОГИЯ РАННЕГО МЕТАБОЛИЧЕСКОГО СИНДРОМА:

T2 - ПАТОГЕНЕТИЧЕСКАЯ РОЛЬ ЮНОШЕСКОГО ДИСПИТУИТАРИЗМА, ДИСПЛАЗИЙ СОЕДИНИТЕЛЬНОЙ ТКАНИ И АУТОИММУННОГО ТИРОИДИТА

AU - Stroev, I. I.

AU - Churilov, L. P.

AU - Agapov, M. M.

AU - Kononova, I. A.

AU - Mudzukova, O. M.

AU - Sadov, S. A.

AU - Serdiuk, I. I.

PY - 2011/7/1

Y1 - 2011/7/1

N2 - A review of world literature, including authors' original papers, establishes the clinical pathophysiological hypothesis, initially coined in by authors in 2002, about regular transition of adolescent hypothalamic syndrome (obesity with rose striae) with age into early metabolic syndrome, complicated by autoimmune thyroiditis. Some evidences are obtained, that witness for marfanoid phenotype and chronic disequilibrium between local, autacoid-mediated and systemic, hormone-mediated regulation, typical for inherited connective tissue disorders, may promote this transition (6 fig., 3 tables, bibl.: 92 ref.).

AB - A review of world literature, including authors' original papers, establishes the clinical pathophysiological hypothesis, initially coined in by authors in 2002, about regular transition of adolescent hypothalamic syndrome (obesity with rose striae) with age into early metabolic syndrome, complicated by autoimmune thyroiditis. Some evidences are obtained, that witness for marfanoid phenotype and chronic disequilibrium between local, autacoid-mediated and systemic, hormone-mediated regulation, typical for inherited connective tissue disorders, may promote this transition (6 fig., 3 tables, bibl.: 92 ref.).

UR - http://www.scopus.com/inward/record.url?scp=84857389964&partnerID=8YFLogxK

M3 - Обзорная статья

C2 - 22279733

AN - SCOPUS:84857389964

SP - 3

EP - 14

JO - ПАТОЛОГИЧЕСКАЯ ФИЗИОЛОГИЯ И ЭКСПЕРИМЕНТАЛЬНАЯ ТЕРАПИЯ

JF - ПАТОЛОГИЧЕСКАЯ ФИЗИОЛОГИЯ И ЭКСПЕРИМЕНТАЛЬНАЯ ТЕРАПИЯ

SN - 0031-2991

IS - 3

ER -

ID: 36150966