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Наследственный панкреатит: обзор. / Балукова, Екатерина Владимировна; Карпеева, Юлия Сергеевна; Новикова, Валерия; Хавкин, Анатолий Ильич.

в: ВОПРОСЫ СОВРЕМЕННОЙ ПЕДИАТРИИ, Том 24, № 1, 25.02.2025, стр. 13-18.

Результаты исследований: Научные публикации в периодических изданияхОбзорная статьяРецензирование

Harvard

Балукова, ЕВ, Карпеева, ЮС, Новикова, В & Хавкин, АИ 2025, 'Наследственный панкреатит: обзор', ВОПРОСЫ СОВРЕМЕННОЙ ПЕДИАТРИИ, Том. 24, № 1, стр. 13-18. https://doi.org/10.15690/vsp.v24i1.2861

APA

Балукова, Е. В., Карпеева, Ю. С., Новикова, В., & Хавкин, А. И. (2025). Наследственный панкреатит: обзор. ВОПРОСЫ СОВРЕМЕННОЙ ПЕДИАТРИИ, 24(1), 13-18. https://doi.org/10.15690/vsp.v24i1.2861

Vancouver

Балукова ЕВ, Карпеева ЮС, Новикова В, Хавкин АИ. Наследственный панкреатит: обзор. ВОПРОСЫ СОВРЕМЕННОЙ ПЕДИАТРИИ. 2025 Февр. 25;24(1):13-18. https://doi.org/10.15690/vsp.v24i1.2861

Author

Балукова, Екатерина Владимировна ; Карпеева, Юлия Сергеевна ; Новикова, Валерия ; Хавкин, Анатолий Ильич. / Наследственный панкреатит: обзор. в: ВОПРОСЫ СОВРЕМЕННОЙ ПЕДИАТРИИ. 2025 ; Том 24, № 1. стр. 13-18.

BibTeX

@article{b2445ec6267e44cc82ecc5957718980d,
title = "Наследственный панкреатит: обзор",
abstract = "Pancreatitis refers to digestive system diseases that are challenging to diagnose and manage. They are also associated with irreversible changes in the organ's parenchyma, that can lead to lethal outcome. The etiology of pancreatitis is diverse due to various exogenous and endogenous factors that determine the nature and severity of the pathological process itself. The major causes in children are the following: obstruction of ductus choledochus or duodenal papilla, abdominal trauma, viral infections, and some medications. Experts have revealed, after several studies, the association between pancreatitis and genetic factors causing up ton 75% of cases of chronic pancreatitis. Nowadays, genes associated with hereditary pancreatitis include PRSS1 (cationic trypsinogen gene), SPINK1 (serine peptidase inhibitor Kazal, type 1), CFTR (cystic fibrosis transmembrane conduction regulator), CTRC (encoding the functions of chymotrypsin C). The diagnostic criteria for hereditary pancreatitis have been established as for any other pancreatitis. The leading method for visualization of changes in pancreas remains ultrasound (US) examination of abdominal organs, endoscopic US, or magnetic resonance imaging. Molecular genetic testing is advisable for children with manifestations of pancreatitis in addition to recommended basic examinations.",
keywords = "hereditary pancreatitis, major pancreatitis genes, pancreatitis risk factors",
author = "Балукова, {Екатерина Владимировна} and Карпеева, {Юлия Сергеевна} and Валерия Новикова and Хавкин, {Анатолий Ильич}",
year = "2025",
month = feb,
day = "25",
doi = "10.15690/vsp.v24i1.2861",
language = "русский",
volume = "24",
pages = "13--18",
journal = "ВОПРОСЫ СОВРЕМЕННОЙ ПЕДИАТРИИ",
issn = "1682-5527",
publisher = "Publishing House of the Union of Pediatricians",
number = "1",

}

RIS

TY - JOUR

T1 - Наследственный панкреатит: обзор

AU - Балукова, Екатерина Владимировна

AU - Карпеева, Юлия Сергеевна

AU - Новикова, Валерия

AU - Хавкин, Анатолий Ильич

PY - 2025/2/25

Y1 - 2025/2/25

N2 - Pancreatitis refers to digestive system diseases that are challenging to diagnose and manage. They are also associated with irreversible changes in the organ's parenchyma, that can lead to lethal outcome. The etiology of pancreatitis is diverse due to various exogenous and endogenous factors that determine the nature and severity of the pathological process itself. The major causes in children are the following: obstruction of ductus choledochus or duodenal papilla, abdominal trauma, viral infections, and some medications. Experts have revealed, after several studies, the association between pancreatitis and genetic factors causing up ton 75% of cases of chronic pancreatitis. Nowadays, genes associated with hereditary pancreatitis include PRSS1 (cationic trypsinogen gene), SPINK1 (serine peptidase inhibitor Kazal, type 1), CFTR (cystic fibrosis transmembrane conduction regulator), CTRC (encoding the functions of chymotrypsin C). The diagnostic criteria for hereditary pancreatitis have been established as for any other pancreatitis. The leading method for visualization of changes in pancreas remains ultrasound (US) examination of abdominal organs, endoscopic US, or magnetic resonance imaging. Molecular genetic testing is advisable for children with manifestations of pancreatitis in addition to recommended basic examinations.

AB - Pancreatitis refers to digestive system diseases that are challenging to diagnose and manage. They are also associated with irreversible changes in the organ's parenchyma, that can lead to lethal outcome. The etiology of pancreatitis is diverse due to various exogenous and endogenous factors that determine the nature and severity of the pathological process itself. The major causes in children are the following: obstruction of ductus choledochus or duodenal papilla, abdominal trauma, viral infections, and some medications. Experts have revealed, after several studies, the association between pancreatitis and genetic factors causing up ton 75% of cases of chronic pancreatitis. Nowadays, genes associated with hereditary pancreatitis include PRSS1 (cationic trypsinogen gene), SPINK1 (serine peptidase inhibitor Kazal, type 1), CFTR (cystic fibrosis transmembrane conduction regulator), CTRC (encoding the functions of chymotrypsin C). The diagnostic criteria for hereditary pancreatitis have been established as for any other pancreatitis. The leading method for visualization of changes in pancreas remains ultrasound (US) examination of abdominal organs, endoscopic US, or magnetic resonance imaging. Molecular genetic testing is advisable for children with manifestations of pancreatitis in addition to recommended basic examinations.

KW - hereditary pancreatitis

KW - major pancreatitis genes

KW - pancreatitis risk factors

UR - https://www.mendeley.com/catalogue/92d81929-7abb-36b3-89c0-9f3861dcf387/

U2 - 10.15690/vsp.v24i1.2861

DO - 10.15690/vsp.v24i1.2861

M3 - Обзорная статья

VL - 24

SP - 13

EP - 18

JO - ВОПРОСЫ СОВРЕМЕННОЙ ПЕДИАТРИИ

JF - ВОПРОСЫ СОВРЕМЕННОЙ ПЕДИАТРИИ

SN - 1682-5527

IS - 1

ER -

ID: 133296150