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Placental changes in type 1 diabetes mellitus. / Zubjitskaya, L. B.; Kosheleva, N. G.; Semenov, V. V.; Nazarova, S. I.; Ailamazyan, E. K.

In: Arkhiv Patologii, Vol. 70, No. 2, 2008, p. 14-17.

Research output: Contribution to journal › Article › peer-review

Harvard

Zubjitskaya, LB, Kosheleva, NG, Semenov, VV, Nazarova, SI & Ailamazyan, EK 2008, 'Placental changes in type 1 diabetes mellitus', Arkhiv Patologii, vol. 70, no. 2, pp. 14-17.

APA

Zubjitskaya, L. B., Kosheleva, N. G., Semenov, V. V., Nazarova, S. I., & Ailamazyan, E. K. (2008). Placental changes in type 1 diabetes mellitus. Arkhiv Patologii, 70(2), 14-17.

Vancouver

Zubjitskaya LB, Kosheleva NG, Semenov VV, Nazarova SI, Ailamazyan EK. Placental changes in type 1 diabetes mellitus. Arkhiv Patologii. 2008;70(2):14-17.

Author

Zubjitskaya, L. B. ; Kosheleva, N. G. ; Semenov, V. V. ; Nazarova, S. I. ; Ailamazyan, E. K. / Placental changes in type 1 diabetes mellitus. In: Arkhiv Patologii. 2008 ; Vol. 70, No. 2. pp. 14-17.

BibTeX

@article{c2d75fdecbf54fe9a577c32b2db2b02e,
title = "Placental changes in type 1 diabetes mellitus",
abstract = "Pathological immune complexes (PIC) are revealed in high percent (100%) of cases by placental immunomorphological studies in insulin-dependent tissue. This leads to the development of an immunopathological process in the placental tissue. Parallel electron microscopy shows that some placental vessels are in close contact with the basal membrane of syncytiotrophoblast. PIC deposition in the area of confluence of the basal membranes of a syncytiotrophoblast and the vascular endothelium, which act as filters, results in the destruction of the basal membranes to rupture formation, causing the transplacental transport of substances together with immune complexes in the form of endocytotic vacuoles from the intervillous lacuna directly to the vessel. Active proliferation of vascular endotheliocyes leads to typical angiopathy as the body's defense reaction.",
author = "Zubjitskaya, {L. B.} and Kosheleva, {N. G.} and Semenov, {V. V.} and Nazarova, {S. I.} and Ailamazyan, {E. K.}",
year = "2008",
language = "English",
volume = "70",
pages = "14--17",
journal = "АРХИВ ПАТОЛОГИИ",
issn = "0004-1955",
publisher = "Медиа Сфера",
number = "2",

}

RIS

TY - JOUR

T1 - Placental changes in type 1 diabetes mellitus

AU - Zubjitskaya, L. B.

AU - Kosheleva, N. G.

AU - Semenov, V. V.

AU - Nazarova, S. I.

AU - Ailamazyan, E. K.

PY - 2008

Y1 - 2008

N2 - Pathological immune complexes (PIC) are revealed in high percent (100%) of cases by placental immunomorphological studies in insulin-dependent tissue. This leads to the development of an immunopathological process in the placental tissue. Parallel electron microscopy shows that some placental vessels are in close contact with the basal membrane of syncytiotrophoblast. PIC deposition in the area of confluence of the basal membranes of a syncytiotrophoblast and the vascular endothelium, which act as filters, results in the destruction of the basal membranes to rupture formation, causing the transplacental transport of substances together with immune complexes in the form of endocytotic vacuoles from the intervillous lacuna directly to the vessel. Active proliferation of vascular endotheliocyes leads to typical angiopathy as the body's defense reaction.

AB - Pathological immune complexes (PIC) are revealed in high percent (100%) of cases by placental immunomorphological studies in insulin-dependent tissue. This leads to the development of an immunopathological process in the placental tissue. Parallel electron microscopy shows that some placental vessels are in close contact with the basal membrane of syncytiotrophoblast. PIC deposition in the area of confluence of the basal membranes of a syncytiotrophoblast and the vascular endothelium, which act as filters, results in the destruction of the basal membranes to rupture formation, causing the transplacental transport of substances together with immune complexes in the form of endocytotic vacuoles from the intervillous lacuna directly to the vessel. Active proliferation of vascular endotheliocyes leads to typical angiopathy as the body's defense reaction.

UR - http://www.scopus.com/inward/record.url?scp=45549104159&partnerID=8YFLogxK

M3 - Article

C2 - 18540434

AN - SCOPUS:45549104159

VL - 70

SP - 14

EP - 17

JO - АРХИВ ПАТОЛОГИИ

JF - АРХИВ ПАТОЛОГИИ

SN - 0004-1955

IS - 2

ER -

ID: 87787290