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Otsinka porushen' kyslotno-luzhnoï rivnovahy v tkanynakh i ridynakh orhanizmu. / Pakhomova, V. O.; Biloklyts'ka, H. F.; Protunkevych, O. O.; Rozanov, V. A.; Pakhomova, O. O.; Hruzova, I. L.; Mel'nychuk, D. O.

In: Fiziolohichnyi zhurnal (Kiev, Ukraine : 1994), Vol. 45, No. 3, 01.01.1999, p. 103-109.

Research output: Contribution to journalArticlepeer-review

Harvard

Pakhomova, VO, Biloklyts'ka, HF, Protunkevych, OO, Rozanov, VA, Pakhomova, OO, Hruzova, IL & Mel'nychuk, DO 1999, 'Otsinka porushen' kyslotno-luzhnoï rivnovahy v tkanynakh i ridynakh orhanizmu.', Fiziolohichnyi zhurnal (Kiev, Ukraine : 1994), vol. 45, no. 3, pp. 103-109.

APA

Pakhomova, V. O., Biloklyts'ka, H. F., Protunkevych, O. O., Rozanov, V. A., Pakhomova, O. O., Hruzova, I. L., & Mel'nychuk, D. O. (1999). Otsinka porushen' kyslotno-luzhnoï rivnovahy v tkanynakh i ridynakh orhanizmu. Fiziolohichnyi zhurnal (Kiev, Ukraine : 1994), 45(3), 103-109.

Vancouver

Pakhomova VO, Biloklyts'ka HF, Protunkevych OO, Rozanov VA, Pakhomova OO, Hruzova IL et al. Otsinka porushen' kyslotno-luzhnoï rivnovahy v tkanynakh i ridynakh orhanizmu. Fiziolohichnyi zhurnal (Kiev, Ukraine : 1994). 1999 Jan 1;45(3):103-109.

Author

Pakhomova, V. O. ; Biloklyts'ka, H. F. ; Protunkevych, O. O. ; Rozanov, V. A. ; Pakhomova, O. O. ; Hruzova, I. L. ; Mel'nychuk, D. O. / Otsinka porushen' kyslotno-luzhnoï rivnovahy v tkanynakh i ridynakh orhanizmu. In: Fiziolohichnyi zhurnal (Kiev, Ukraine : 1994). 1999 ; Vol. 45, No. 3. pp. 103-109.

BibTeX

@article{fcee8d2ab11248d98fb2b9b53ca31dfc,
title = "Otsinka porushen' kyslotno-luzhno{\"i} rivnovahy v tkanynakh i ridynakh orhanizmu.",
abstract = "Ions hydrogene concentration in fluids and tissues is one of the strict regulated physiological organism variables. Most common man and animal diseases are accompanied with compensated acid-base balance shift development. But diagnosis of metabolic acidosis and alkalosis with blood indices (pH, pCO2, HCO3) doesn't reflect true tissue condition that is connected not only with respiratory and excretion system functioning but with hypercompensation of metabolic processes. Excess organic acid formation in case of metabolic alkalosis formation in tissues directed to support pH leads to acidification, what brings an essential mistake in pathogenesis understanding of such diseases as cardiomyopathy, ischemic cardiac disease, myocardial infarction, hepatitis, collagenosis, caries, peptic ulcer, macula dystrophy, atherosclerosis, virus diseases, radiation damages. Ignorance primary tissue damages doesn't allow to conduct necessary preventive measures and to cure disease. It is suggested to assess development of metabolic acidosis and alkalosis by compensated changes of regulation acid-base homeostasis system, which is the basis of all future damages and development of different diseases against this background.",
author = "Pakhomova, {V. O.} and Biloklyts'ka, {H. F.} and Protunkevych, {O. O.} and Rozanov, {V. A.} and Pakhomova, {O. O.} and Hruzova, {I. L.} and Mel'nychuk, {D. O.}",
year = "1999",
month = jan,
day = "1",
language = "украинский",
volume = "45",
pages = "103--109",
journal = "Fiziolohichnyi zhurnal",
issn = "0201-8489",
publisher = "Naukova Dumka",
number = "3",

}

RIS

TY - JOUR

T1 - Otsinka porushen' kyslotno-luzhnoï rivnovahy v tkanynakh i ridynakh orhanizmu.

AU - Pakhomova, V. O.

AU - Biloklyts'ka, H. F.

AU - Protunkevych, O. O.

AU - Rozanov, V. A.

AU - Pakhomova, O. O.

AU - Hruzova, I. L.

AU - Mel'nychuk, D. O.

PY - 1999/1/1

Y1 - 1999/1/1

N2 - Ions hydrogene concentration in fluids and tissues is one of the strict regulated physiological organism variables. Most common man and animal diseases are accompanied with compensated acid-base balance shift development. But diagnosis of metabolic acidosis and alkalosis with blood indices (pH, pCO2, HCO3) doesn't reflect true tissue condition that is connected not only with respiratory and excretion system functioning but with hypercompensation of metabolic processes. Excess organic acid formation in case of metabolic alkalosis formation in tissues directed to support pH leads to acidification, what brings an essential mistake in pathogenesis understanding of such diseases as cardiomyopathy, ischemic cardiac disease, myocardial infarction, hepatitis, collagenosis, caries, peptic ulcer, macula dystrophy, atherosclerosis, virus diseases, radiation damages. Ignorance primary tissue damages doesn't allow to conduct necessary preventive measures and to cure disease. It is suggested to assess development of metabolic acidosis and alkalosis by compensated changes of regulation acid-base homeostasis system, which is the basis of all future damages and development of different diseases against this background.

AB - Ions hydrogene concentration in fluids and tissues is one of the strict regulated physiological organism variables. Most common man and animal diseases are accompanied with compensated acid-base balance shift development. But diagnosis of metabolic acidosis and alkalosis with blood indices (pH, pCO2, HCO3) doesn't reflect true tissue condition that is connected not only with respiratory and excretion system functioning but with hypercompensation of metabolic processes. Excess organic acid formation in case of metabolic alkalosis formation in tissues directed to support pH leads to acidification, what brings an essential mistake in pathogenesis understanding of such diseases as cardiomyopathy, ischemic cardiac disease, myocardial infarction, hepatitis, collagenosis, caries, peptic ulcer, macula dystrophy, atherosclerosis, virus diseases, radiation damages. Ignorance primary tissue damages doesn't allow to conduct necessary preventive measures and to cure disease. It is suggested to assess development of metabolic acidosis and alkalosis by compensated changes of regulation acid-base homeostasis system, which is the basis of all future damages and development of different diseases against this background.

UR - http://www.scopus.com/inward/record.url?scp=0032608958&partnerID=8YFLogxK

M3 - статья

C2 - 10439299

AN - SCOPUS:0032608958

VL - 45

SP - 103

EP - 109

JO - Fiziolohichnyi zhurnal

JF - Fiziolohichnyi zhurnal

SN - 0201-8489

IS - 3

ER -

ID: 42768413