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Influenza, autoimmunity and atherogenesis. / Gurevich, VS.

In: Autoimmunity Reviews, Vol. 4, No. 2, 2005, p. 101-105.

Research output: Contribution to journal › Review article › peer-review

Harvard

Gurevich, VS 2005, 'Influenza, autoimmunity and atherogenesis', Autoimmunity Reviews, vol. 4, no. 2, pp. 101-105. https://doi.org/10.1016/j.autrev.2004.10.006

APA

Gurevich, VS. (2005). Influenza, autoimmunity and atherogenesis. Autoimmunity Reviews, 4(2), 101-105. https://doi.org/10.1016/j.autrev.2004.10.006

Vancouver

Gurevich VS. Influenza, autoimmunity and atherogenesis. Autoimmunity Reviews. 2005;4(2):101-105. https://doi.org/10.1016/j.autrev.2004.10.006

Author

Gurevich, VS. / Influenza, autoimmunity and atherogenesis. In: Autoimmunity Reviews. 2005 ; Vol. 4, No. 2. pp. 101-105.

BibTeX

@article{34e2904801f241ee8e449922ffcf9e59,
title = "Influenza, autoimmunity and atherogenesis",
abstract = "It has been observed during influenza epidemics and in a number of population and clinical trials that this prevalent viral infection was associated with increased death rates from cardiovascular diseases. The clinical and experimental data that may explain accelerated coronary atherosclerosis in influenza infection with implications involving autoimmune mechanisms are reviewed in this article. Both cellular and humoral autoimmune mode could be proposed to participate in the onset or progression of atheromatous lesions due to influenza infection.",
keywords = "Animals, Autoantibodies/immunology, Autoimmunity, Coronary Artery Disease/etiology, Humans, Influenza Vaccines/administration & dosage, Influenza, Human/complications, Lipid Peroxidation, Lipoproteins, LDL/immunology, Molecular Mimicry",
author = "VS Gurevich",
year = "2005",
doi = "10.1016/j.autrev.2004.10.006",
language = "English",
volume = "4",
pages = "101--105",
journal = "Autoimmunity Reviews",
issn = "1568-9972",
publisher = "Elsevier",
number = "2",

}

RIS

TY - JOUR

T1 - Influenza, autoimmunity and atherogenesis

AU - Gurevich, VS

PY - 2005

Y1 - 2005

N2 - It has been observed during influenza epidemics and in a number of population and clinical trials that this prevalent viral infection was associated with increased death rates from cardiovascular diseases. The clinical and experimental data that may explain accelerated coronary atherosclerosis in influenza infection with implications involving autoimmune mechanisms are reviewed in this article. Both cellular and humoral autoimmune mode could be proposed to participate in the onset or progression of atheromatous lesions due to influenza infection.

AB - It has been observed during influenza epidemics and in a number of population and clinical trials that this prevalent viral infection was associated with increased death rates from cardiovascular diseases. The clinical and experimental data that may explain accelerated coronary atherosclerosis in influenza infection with implications involving autoimmune mechanisms are reviewed in this article. Both cellular and humoral autoimmune mode could be proposed to participate in the onset or progression of atheromatous lesions due to influenza infection.

KW - Animals

KW - Autoantibodies/immunology

KW - Autoimmunity

KW - Coronary Artery Disease/etiology

KW - Humans

KW - Influenza Vaccines/administration & dosage

KW - Influenza, Human/complications

KW - Lipid Peroxidation

KW - Lipoproteins, LDL/immunology

KW - Molecular Mimicry

U2 - 10.1016/j.autrev.2004.10.006

DO - 10.1016/j.autrev.2004.10.006

M3 - Review article

C2 - 15722256

VL - 4

SP - 101

EP - 105

JO - Autoimmunity Reviews

JF - Autoimmunity Reviews

SN - 1568-9972

IS - 2

ER -

ID: 5459799