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Chaperone effects on prion and nonprion aggregates. / Rikhvanov, Eugene G.; Romanova, Nina V.; Chernoff, Yury O.

Protein-Based Inheritance. Taylor & Francis, 2007. p. 83-92.

Research output: Chapter in Book/Report/Conference proceedingChapterResearchpeer-review

Harvard

Rikhvanov, EG, Romanova, NV & Chernoff, YO 2007, Chaperone effects on prion and nonprion aggregates. in Protein-Based Inheritance. Taylor & Francis, pp. 83-92. https://doi.org/10.1201/9781498712507-13

APA

Rikhvanov, E. G., Romanova, N. V., & Chernoff, Y. O. (2007). Chaperone effects on prion and nonprion aggregates. In Protein-Based Inheritance (pp. 83-92). Taylor & Francis. https://doi.org/10.1201/9781498712507-13

Vancouver

Rikhvanov EG, Romanova NV, Chernoff YO. Chaperone effects on prion and nonprion aggregates. In Protein-Based Inheritance. Taylor & Francis. 2007. p. 83-92 https://doi.org/10.1201/9781498712507-13

Author

Rikhvanov, Eugene G. ; Romanova, Nina V. ; Chernoff, Yury O. / Chaperone effects on prion and nonprion aggregates. Protein-Based Inheritance. Taylor & Francis, 2007. pp. 83-92

BibTeX

@inbook{58b37afb4b544b969832d1cfde0e560f,
title = "Chaperone effects on prion and nonprion aggregates",
abstract = "Exposure to high temperature or other stresses induces a synthesis of heat shock proteins. Many of these proteins are molecular chaperones, and some of them help cells to cope with heat-induced denaturation and aggregation of other proteins. In the last decade, chaperones have received increased attention in connection with their role in maintenance and propagation of the Saccharomyces cerevisiae prions, infectious or heritable agents transmitted at the protein level. Recent data suggest that functioning of the chaperones in reactivation of heat-damaged proteins and in propagation of prions is based on the same molecular mechanisms but may lead to different consequences depending on the type of aggregate. In both cases the conceited and balanced action of “chaperones{\textquoteright} team”, including Hsp 104, Hsp70, Hsp40 and possibly other proteins, determines whether a misfolded protein is to be incorporated into an aggregate, rescued to the native state or targeted for degradation.",
author = "Rikhvanov, {Eugene G.} and Romanova, {Nina V.} and Chernoff, {Yury O.}",
year = "2007",
month = jan,
day = "1",
doi = "10.1201/9781498712507-13",
language = "English",
isbn = "9781587061387",
pages = "83--92",
booktitle = "Protein-Based Inheritance",
publisher = "Taylor & Francis",
address = "United Kingdom",

}

RIS

TY - CHAP

T1 - Chaperone effects on prion and nonprion aggregates

AU - Rikhvanov, Eugene G.

AU - Romanova, Nina V.

AU - Chernoff, Yury O.

PY - 2007/1/1

Y1 - 2007/1/1

N2 - Exposure to high temperature or other stresses induces a synthesis of heat shock proteins. Many of these proteins are molecular chaperones, and some of them help cells to cope with heat-induced denaturation and aggregation of other proteins. In the last decade, chaperones have received increased attention in connection with their role in maintenance and propagation of the Saccharomyces cerevisiae prions, infectious or heritable agents transmitted at the protein level. Recent data suggest that functioning of the chaperones in reactivation of heat-damaged proteins and in propagation of prions is based on the same molecular mechanisms but may lead to different consequences depending on the type of aggregate. In both cases the conceited and balanced action of “chaperones’ team”, including Hsp 104, Hsp70, Hsp40 and possibly other proteins, determines whether a misfolded protein is to be incorporated into an aggregate, rescued to the native state or targeted for degradation.

AB - Exposure to high temperature or other stresses induces a synthesis of heat shock proteins. Many of these proteins are molecular chaperones, and some of them help cells to cope with heat-induced denaturation and aggregation of other proteins. In the last decade, chaperones have received increased attention in connection with their role in maintenance and propagation of the Saccharomyces cerevisiae prions, infectious or heritable agents transmitted at the protein level. Recent data suggest that functioning of the chaperones in reactivation of heat-damaged proteins and in propagation of prions is based on the same molecular mechanisms but may lead to different consequences depending on the type of aggregate. In both cases the conceited and balanced action of “chaperones’ team”, including Hsp 104, Hsp70, Hsp40 and possibly other proteins, determines whether a misfolded protein is to be incorporated into an aggregate, rescued to the native state or targeted for degradation.

UR - http://www.scopus.com/inward/record.url?scp=77952019194&partnerID=8YFLogxK

U2 - 10.1201/9781498712507-13

DO - 10.1201/9781498712507-13

M3 - Chapter

SN - 9781587061387

SP - 83

EP - 92

BT - Protein-Based Inheritance

PB - Taylor & Francis

ER -

ID: 49545569