1. 2019
  2. Hypoxia as a Factor Involved in the Regulation of the apoA-1, ABCA1, and Complement C3 Gene Expression in Human Macrophages

    Bogomolova, A. M., Shavva, V. S., Nikitin, A. A., Nekrasova, E. V., Dizhe, E. B., Larionova, E. E., Kudriavtsev, I. V. & Orlov, S. V., 1 May 2019, In: Biochemistry (Moscow). 84, 5, p. 529-539 11 p.

    Research output: Contribution to journalArticlepeer-review

  3. Влияние крамизола на экспрессию гена аполипопротеина А1 (АпоА1) в печени крыс при экспериментально индуцированной гиперлипидемии.

    Lizunov, A. V., Okunevich, I. V., Orlov, S. V., Lebedev, A. A., Bychkov, E. R., Piotrovskiy, L. B. & Shabanov, P. D., 2019, In: БИОМЕДИЦИНСКАЯ ХИМИЯ. 65, 5, p. 403-406 4 p.

    Research output: Contribution to journalArticlepeer-review

  4. Характеристика альтернативных промоторов гена аполипопротеина A-I человека.

    Могиленко, Д. А., Шавва, В. С., Диже, Э. Б. & Орлов, С. В., 2019, In: МОЛЕКУЛЯРНАЯ БИОЛОГИЯ. 53, 3, p. 485-496

    Research output: Contribution to journalArticlepeer-review

  5. 2018
  6. Tumor necrosis factor α stimulates endogenous apolipoprotein A-I expression and secretion by human monocytes and macrophages: role of MAP-kinases, NF-κB, and nuclear receptors PPARα and LXRs

    Shavva, V. S., Mogilenko, D. A., Nekrasova, E. V., Trulioff, A. S., Kudriavtsev, I. V., Larionova, E. E., Babina, A. V., Dizhe, E. B., Missyul, B. V. & Orlov, S. V., 1 Nov 2018, In: Molecular and Cellular Biochemistry. 448, 1-2, p. 211-223 13 p.

    Research output: Contribution to journalArticlepeer-review

  7. Insulin downregulates C3 gene expression in human HepG2 cells through activation of PPARγ

    Shavva, V. S., Bogomolova, A. M., Efremov, A. M., Trofimov, A. N., Nikitin, A. A., Babina, A. V., Nekrasova, E. V., Dizhe, E. B., Oleinikova, G. N., Missyul, B. V. & Orlov, S. V., 1 Apr 2018, In: European Journal of Cell Biology. 97, 3, p. 204-215 12 p.

    Research output: Contribution to journalArticlepeer-review

  8. 2017
  9. Insulin-Mediated Downregulation of Apolipoprotein A-I Gene in Human Hepatoma Cell Line HepG2: The Role of Interaction Between FOXO1 and LXR beta Transcription Factors

    Shavva, V. S., Bogomolova, A. M., Nikitin, A. A., Dizhe, E. B., Tanyanskiy, D. A., Efremov, A. M., Oleinikova, G. N., Perevozchikov, A. P. & Orlov, S. V., Feb 2017, In: Journal of Cellular Biochemistry. 118, 2, p. 382-396 15 p.

    Research output: Contribution to journalArticlepeer-review

  10. FOXO1 and LXR alpha downregulate the apolipoprotein A-I gene expression during hydrogen peroxide-induced oxidative stress in HepG2 cells

    Shavva, V. S., Bogomolova, A. M., Nikitin, A. A., Dizhe, E. B., Oleinikova, G. N., Lapikov, I. A., Tanyanskiy, D. A., Perevozchikov, A. P. & Orlov, S. V., Jan 2017, In: Cell Stress and Chaperones. 22, 1, p. 123-134 12 p.

    Research output: Contribution to journalArticlepeer-review

  11. 2016
  12. PPARγ represses apolipoprotein A-I gene but impedes TNFα-mediated ApoA-I downregulation in HepG2 cells

    Shavva, V. S., Mogilenko, D. A., Bogomolova, A. M., Nikitin, A. A., Dizhe, E. B., Efremov, A. M., Oleinikova, G. N., Perevozchikov, A. P. & Orlov, S. V., 2016, In: Journal of Cellular Biochemistry. 117, 9, p. 2010-2022

    Research output: Contribution to journalArticlepeer-review

  13. 2013
  14. Hepatic nuclear factor 4α positively regulates complement C3 expression and does not interfere with TNFα-mediated stimulation of C3 expression in HepG2 cells

    Shavva, V. S., Mogilenko, D. A., Dizhe, E. B., Oleinikova, G. N., Perevozchikov, A. P. & Orlov, S. V., 2013, In: Gene. 524, 2, p. 187-192 6 p.

    Research output: Contribution to journalArticle

  15. Peroxisome Proliferator-activated Receptor α Positively Regulates Complement C3 Expression but Inhibits Tumor Necrosis Factor α-mediated Activation of C3 Gene in Mammalian Hepatic-derived Cells

    Mogilenko, D. A., Kudriavtsev, I. V., Shavva, V. S., Dizhe, E. B., Vilenskaya, E. G., Efremov, A. M., Perevozchikov, A. P. & Orlov, S. V., 2013, In: Journal of Biological Chemistry. 288, 3, p. 1726-1738 13 p.

    Research output: Contribution to journalArticle

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